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中国科学院上海生命科学研究院时玉舫任国际生物医学重要学术期刊《Cell Death & Disease》主编
时玉舫 国际生物医学 重要学术期刊 Cell Death & Disease 主编
2013/2/21
国际生物医学重要学术期刊《Cell Death & Disease》已任命中国科学院上海生命科学研究院/上海交通大学医学院健康科学研究所所长时玉舫为主编。《Cell Death & Disease》系《Nature》出版集团（Nature Publication Group，NPG）属下的权威医学生命科学期刊之一，主要关注转化医学研究，特别是癌症、免疫学、神经学等领域的高水平、原创性研究发展。《C...
Aged Garlic Extract Reduces ROS Production and Cell Death Induced by 6-Hydroxydopamine through Activation of the Nrf2-ARE Pathway in SH-SY5Y Cells
Aged Garlic Extract (AGE) 6-OHDA ROS Nrf2-ARE Pathway SH-SY5Y Cells
2013/2/19
Many degenerative or pathological processes, such as aging, cancer and coronary heart disease, are related to reactive oxygen species (ROS) and radical-mediated reactions. We examined the effectivenes...
Reactive oxygen species induced oxidative stress, neuronal apoptosis and alternative death pathways
Apoptosis Caspases Cell Death Mitochondria PCBs Reactive Oxygen Species
2013/1/28
Reactive oxygen species (ROS) are produced as a byproduct of cellular metabolic pathways and function as a critical second messenger in a variety of intracellular signaling pathways. The excessive int...
Engineered FADD Induces Apoptosis via an Artificial Death-Inducing Signaling Complex (DISC)
apoptosis FADD biotechnology DISC engineered protein
2009/10/26
An engineered Fas-associated death domain protein (FADD), 2DEDplusE—made previously by fusing the tandem DEDs of FADD to the E protein of lambda phage-greatly enhances apoptosis-inducing activity in a...
New Advance in Caspase-Independent Programmed Cell Death and Its Potential in Cancer Therapy
apoptosis autophagy cancer therapy caspase-independent programmed cell death
2009/4/20
Caspase activation has been frequently viewed as synonymous with programmed cell death (PCcD); however, accumulating evidence showed that there existing caspase-independent PCcD pathways displaying m...
Involvement of Zinc in Neuronal Death in the Hippocampus
zinc glutamate excitotoxicity hippocampus calcium
2009/3/16
Zinc is released with glutamate from neuron terminals in the hippocampus. Zinc may serve as a negative-feedback factor of presynaptic activity and negatively modulate postsynaptic calcium mobilization...